Synopsis

In the late 20th century, a devastating disease emerged within British cattle herds, later termed Mad Cow disease. As the illness spread, scientists began to ponder its connection to human ailments. A neurologist in California posited that both species were afflicted by a mutated prion. These defective proteins, when transferred from an infected animal to a new host, would trigger normal prions to replicate into their altered form, suggesting a possible route of transmission through consumption